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demonstrated for the first time that TMAO can bind and activate PERK to cause atherosclerosis, which provides a new mechanism of action of TMAO-induced atherosclerosis, which will be described in detail below ( Effect of TMAO and Its Precursor (L-CARNITINE) on Atherosclerosis TMAO and Atherosclerosis Atherosclerosis is the pathological basis of coronary atherosclerotic heart disease ( TMAO and Chronic Coronary Artery Disease In recent years, several studies have suggested that TMAO is a new independent risk factor for atherosclerosis (Table 1)

The present study also explores the possibility that the oxidized inactive form of e-GST may be present in blood, given that two critical cysteines, Cys47 and Cys101 (the latter corresponding to Cys103 in pig e-GST) are strictly conserved in all mammals tested, and that they form a disulfide both in vivo and in vitro as proved for the human enzyme

Mazilescu, L

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Rudolf Weber, Agency for Scientific Publications, 2002)