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doi: 10.1016/j.tox.2008.01.011

It does not diagnose, treat, or recommend medical use

In contrast, the present study targets the early signaling event of NETosis, namely mtROS accumulation, and utilizes mitochondrial-targeted TPP-CeO 2 in combination with the anti-inflammatory effects of Res to effectively suppress CitH3 and MPO expression, thereby achieving multi-level inhibition of NETosis

This view is consistent with arterio-venous difference studies showing that the brain is a net exporter of free choline, with the rate of choline efflux varying depending on metabolic conditions [70, 79]

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