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Additionally, osteocytes display impaired perilacunar/canalicular remodeling and diminished mechanosensing capacity, further disrupting local bone homeostasis (Zhu et al., 2020

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8a, b, provides a plausible alternative mechanism for the reduction of non-glutathione disulfide substrates that could complement the established dithiol mechanism for EcGrx1 and EcRNR

4b, c, Supplementary Fig